FOXO1 | Forkhead box protein O1
 
image/svg+xmlExtracellular space Cytosol Plasma membrane Cytoskeleton Lysosome Endosome Peroxisome ER Golgi Apparatus Nucleus Mitochondrion None Substrate Localization legend

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Sequence Viewer
Gene
Synonyms
FKHR FOXO1A
Protein Name
Forkhead box protein O1
UniProt ID
Q12778 [go to UniProt ] [go to PDBe-KB ]
Ensembl Gene ID
NCBI Gene ID
Molecular Weight
69662
Protein Length
655
Protein Domain
3D Structure
(PDB ID : 3co6)
Target by Small Molecules
Protein-protein Interaction Database
Gene Expression
Drugs and Diseases
Enzyme Class
-
Catalytic Site
Catalytic Activity
-
Localization
Cytoplasm,Nucleus;
Function
Transcription factor that is the main target of insulin signaling and regulates metabolic homeostasis in response to oxidative stress. Binds to the insulin response element (IRE) with consensus sequence 5'-TT[G/A]TTTTG-3' and the related Daf-16 family binding element (DBE) with consensus sequence 5'-TT[G/A]TTTAC-3'. Activity suppressed by insulin. Main regulator of redox balance and osteoblast numbers and controls bone mass. Orchestrates the endocrine function of the skeleton in regulating glucose metabolism. Acts synergistically with ATF4 to suppress osteocalcin/BGLAP activity, increasing glucose levels and triggering glucose intolerance and insulin insensitivity. Also suppresses the transcriptional activity of RUNX2, an upstream activator of osteocalcin/BGLAP. In hepatocytes, promotes gluconeogenesis by acting together with PPARGC1A and CEBPA to activate the expression of genes such as IGFBP1, G6PC and PCK1. Important regulator of cell death acting downstream of CDK1, PKB/AKT1 and STK4/MST1. Promotes neural cell death. Mediates insulin action on adipose tissue. Regulates the expression of adipogenic genes such as PPARG during preadipocyte differentiation and, adipocyte size and adipose tissue-specific gene expression in response to excessive calorie intake. Regulates the transcriptional activity of GADD45A and repair of nitric oxide-damaged DNA in beta-cells. Required for the autophagic cell death induction in response to starvation or oxidative stress in a transcription-independent manner. Mediates the function of MLIP in cardiomyocytes hypertrophy and cardiac remodeling (By similarity).
Gene Ontology
GO:0005737; GO:0005829; GO:0005739; GO:0005654; GO:0005634; GO:0008013; GO:0003682; GO:0003700; GO:0000981; GO:0001227; GO:0051721; GO:0043565; GO:0001223; GO:0008134; GO:0031625; GO:0006915; GO:0006914; GO:0001568; GO:0001678; GO:0070417; GO:0071549; GO:0006974; GO:0070301; GO:0071455; GO:0032869; GO:0071732; GO:0034599; GO:0009267; GO:0019221; GO:0070166; GO:0031018; GO:0097009; GO:0045444; GO:0042593; GO:0008286; GO:0043066; GO:0090090; GO:1903243; GO:0045599; GO:0032873; GO:0000122; GO:0045892; GO:0097150; GO:0043065; GO:0010508; GO:0045722; GO:0045732; GO:0045944; GO:0045893; GO:0006473; GO:0035947; GO:2000177; GO:2000377; GO:1902617; GO:0001659
 
Gene Ontology